Powerlight BV

Wassercluster sind unsere Spezialität

For Afrika

 

Here you also find  the speech on the occasion of the

"Congress Prevencao e Cura" 21.Dec.2016  Lisbon

 Lecture   

MALARIA 

Malaria is a tropical desease caused by eucaryotic parasites of the genus plasmodium desease vector is the female anopheles mosquito. Following pathogens are considered to be as human pathogen:

- Plasmodium faciparum -- malaria tropica

- Plasmodium vivax -- malaria tertiana

- Plasmodium malariae -- malaria quartana

- Plasmodium knowlesi -- strain from Southeast Asia

There are about 1,5 million victims in the world

The mosquito needs plasmodium as an intermidiate host and vertrebrates or humans as main hosts, to accomplish their life cycle.

Malaria symtomps are high, recurring fever, chills, cramps and gastrointerstinal disorders. Especially for children this disease can be fatal. It is a risk for pregnant women, too.

Residents of regions that are endangered by malaria may develop a partially immunity and get not ill or only a softer way. But these persons are also carrier of the various plasmodium species. That often leads to recurring periods of difficulties in concentration and forgetfulness, because in these patients vessels in the central nervous are affected (also these cases can be healed with Powerlight MA)

After infection the pathogen needs 2-3 days to grow up in erythrocytes. Most patients improve in this phase, when the growth in the red blood cells happens. Onwards the condition deteriods when the erythrocytes burst. This is the reason why the illness was called "agues". In some people living in the malaria regions in the erytrocytes develope different kinds of haemoglobin mutations, leading to sickle cell anaemia in tropical Africa. Thalassemia emerges in this context in the mediterranean region, in Northafrica and South Asia. The glucose-6-phosphate dehydrogenase deficiency is one of changes by mutation of the erythrocytes. In the following malaria patients get an increasing weakening of the body.

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Infection

The live cycle of plasmodium can be classified in 3 phases:

- the exo - erythrocytic phase

- the erythrocytic phase

- phase in the Anopheles moscito.

The infections begins after the bite of the Anopheles mosquito by the sporozoites in the saliva of Anopheles. Sporozoites are small mobil form of life of the plasmodium. They move into the blood vessels and are transported to the liver, where they reproduce multiply. This phase for the host organism is without symptoms and determinds the incubation. Depending on the plasmodium strain the incubation period is between 12 and 50 days.

For some months a part of the sporozoites remains in the liver in a resting phase, to reproduce themselves later. In this resting phase the pathogen is a hypnozoit. Because of this hypnozoit stage, in malaria patients relapses occur again and again. In the liver sporozoites develop, where they grow to schizontes. In there thousands of daughter cells develop (merozoites). When schizontes burst, merozoites invade into the bloodstream. Here the erythrocytic phase begins. Merozoites invade into erythrozytes, ripent there to schizontes and split by cell division. The new merozoites attack other red blood cells. After some of these cycles a part of the plasmodium develops into long living male and female gametes (gametozytes). In the stomach of the moscito the male and female gametozytes merge to ookinetes (mobile zygote). They enter into the interstinal wall, there they form myriads of sporozoites. Sporozoites penetrate the interstinal wall and get into the salivary gland of the moscito. When the moscito stitches again the cycle starts afresh.

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Effect of Powerlight MA

The surface of plasmodium shows proteins connecting with receptors of the surface of erythrocites - then the erythrocite is infected. One of these proteins called PfRh5 reacting with a receptor on the erythrocites, by the cluster of Powerlight MA is electrically changed, so that the connection between PfPh5 and Erythrocytes is not possible. In the same time Powerlight MA makes it possible that the plasmodium can be attacked by the immune system - parts of the cell wall dissolve and can be exreated. Phagocytes remove the rest of the plasmodium.




HIV 

Aids is an acquired illness of the immune system. Pathogen is the HI - Virus. Every year 2 Million people die of AIDS. The disease developes in three stages:

Stage 1: HIV positive - without symptoms

Stage 2: HIV positive - with symptoms

Stage 3: full blown AIDS

Even when we talk about "symptoms free status" in stage 1 there is exhaustion, indisposition, inefficiency, fever, joint- and headache, increased lymph nodes and splenomegaly. Clinically we see large quantities of HI-viruses in the genital secretion. All these symptoms disappear after some time.

The individual process is very different - in most cases several years pass before additionally symptoms occur. In stage 2 there is fever over 38,5 degrees of Celsius that stays longer than one month. Diarrhoea occurs, too. Candidoses in mouth and pharynx also in areas of femal genitales and herpes zoster in different regions of the body also belong to stage 2. In arms and legs there are neuropathies. In the cervix to tissue´s transformations. Aditionally oral Leukoplakia may occure. It comes to infections transmitted by animals, also we see abcesses in fallopian tube or ovary.

In the stage 3 we see the full blown AIDS. Significant loss of weight, extreme tiredness, cough, shortness of breath, intense headache, nausea, impairments of brain functions and intellectuall losses, psycic changes, forgetfullness, confusion, reduced eyesight. It comes to more infections by viruses, bacteria, parasites and fungus infestation. Tubercolosis, toxoplasmosis, salmonella and lung diseases may add. It comes to coma and death.